[1]程勇,张谢夫#,赵春临,等.胃癌组织中SOCS3与STAT3 mRNA的表达[J].郑州大学学报(医学版),2011,(03):468.
 [J].JOURNAL OF ZHENGZHOU UNIVERSITY(MEDICAL SCIENCES),2011,(03):468.
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胃癌组织中SOCS3与STAT3 mRNA的表达()
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《郑州大学学报(医学版)》[ISSN:1671-6825/CN:41-1340/R]

卷:
期数:
2011年03期
页码:
468
栏目:
应用研究
出版日期:
2011-05-15

文章信息/Info

作者:
 程勇张谢夫#赵春临蒋志强
 郑州大学第一附属医院胃肠外科 郑州 450052
关键词:
胃癌细胞信号传导抑制因子3信号传导和转录激活因子3
文献标志码:
A

参考文献/References:

[1]Huang WT,Yang SF,Wu CC,et al.Expression of signal transducer and activator of transcription 3 and suppressor of cytokine signaling 3 in urothelial carcinoma[J].Kaohsiung J Med Sci,2009,25(12):640
[2]Ehrmann J,Strakova N,Vrzalikova K,et al.Expression of STATs and their inhibitors SOCS and PIAS in brain tumors[J]. Neoplasma,2008,55(6):482
[3]田志刚. JAK/STAT信号传导通路特异抑制物SOCS 系统的发现及意义[J].中国免疫学杂志,2000,16(8):454
[4]刘艳明,农光民,李树全.支气管哮喘小鼠呼吸道上皮细胞信号转导与转录活化因子3的表达及其在呼吸道重塑中的作用[J].实用儿科临床杂志,2010,25(4):242
[5]赵淑华,赵凡,郑晶莹,等.RNAi沉默STAT3基因对人卵巢癌SKOV3细胞裸鼠移植瘤生长的抑制作用[J].吉林大学学报:医学版,2010,36(4):640
[6]马洪涛,史艳平,封全灵.卵巢上皮性癌组织中磷酸化信号转导及转录活化因子3和Ki67蛋白的表达[J].郑州大学学报:医学版,2010,45(2):246
[7]吴民华,陈小毅,陈东.鼻咽癌组织中信号转导与转录活化因子3与潜伏膜蛋白1的表达及其关系[J].西安交通大学学报:医学版,2010,31(5):634
[8]Lu Y,Fukuyama S,Yoshida R,et al.Loss of SOCS3 gene expression converts STAT3 function from antiapoptotic to proapoptotic[J].J Biol Chem,2006,281(48):36683
[9]Alvarez JV,Frank DA.Genomewide analysis of STAT target genes: elucidating the mechanism of STATmediated oncogenesis[J]. Cancer Biol Ther,2004,3(11):1045
[10]Yoshimura A. Signal transduction of inflammatory cytokines and tumor development[J].Cancer Sci,2006,97(6):439
[11]Howard JK, Cave BJ, Oksanen LJ, et al. Enhanced leptin senxitivity and attenuation of dietinduced obesity in mice with haploinsufficiency of Socs3[J].Nat Med,2004,10(7):734
[12]Hodge DR,Hurt EM,Farrar WL. The role of IL6 and STAT3 in inflammation and cancer[J].Eur J Cancer,2005,41(16):2502
[13]He B,You L,Uematsu K,et al.SOCS3 is frequently silenced by hypermethylation and suppresses cell growth in human lung cancer[J].Proc Natl Acad Sci USA,2003,100(24):14133
[14]Niwa Y,Kanda H,Shikauchi Y,et al.Methylation silencing of SOCS3 promotes cell growth and migration by enhancing JAK/STAT and FAK signalings in human hepatocellular carcinoma[J].Oncogene,2005,24(42):6406
[15]Weber A,Hengge UR,Bardenheuer W,et al.SOCS3 is frequently methylated in head and neck squamous cell carcinoma and its precursor lesions and causes growth inhibition[J].Oncogene,2005,24(44):6699

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备注/Memo

备注/Memo:
 #通讯作者,男,1957年3月生,本科,教授,研究方向:普外科基础与临床, E-mail:zhangxiefu@medmail.com.cn
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